Semax (10mg)
Spécifications techniques
| Pureté (HPLC) | ≥99% (HPLC) |
| Numéro CAS | 80714-61-0 |
| Forme | White lyophilized powder |
| Stockage | -20°C |
| Séquence | Met-Glu-His-Phe-Pro-Gly-Pro |
| Poids moléculaire | 813.93 g/mol |
| Forme saline | Acetate |
| Solubilité | Soluble in water |
Semax (10mg)
Prix catalogue EUR · HU, SK, CZ, PL
- Eurozone (EUR)83,26 EUR
- Hongrie (HU)83,26 EUR≈ 33 304 Ft
- Slovaquie (SK)83,26 EUR
- Tchéquie (CZ)83,26 EUR≈ 2 082 Kč
- Pologne (PL)83,26 EUR≈ 353,86 zł
Même prix catalogue en EUR pour livraison vers la Hongrie, la Slovaquie, la République tchèque et la Pologne. Paiement en EUR.
Montants HUF/CZK/PLN indicatifs convertis depuis l’EUR ; la Slovaquie utilise l’EUR. Taux approximatifs. Paiement en EUR.
ACTH-derived nootropic peptide for cognitive research.
Mechanism: Increases BDNF, modulates dopamine/serotonin, antioxidant protection.
Applications: Ischemic stroke, cognitive enhancement, neuroprotection.
Protocol: Intranasal delivery typical. Reconstitute 1ml BAC water.
Strictement réservé à la recherche en laboratoire.
Non destiné à la consommation humaine, vétérinaire ou au diagnostic.
From the Peptide Explorer
A heptapeptide derived from the ACTH(4-10) fragment, developed at the Institute of Molecular Genetics. Unlike the parent ACTH molecule, Semax lacks corticotropic and steroidogenic activity, instead focusing on nootropic and neuroprotective pathways.
Key Mechanisms
- ›BDNF & NGF upregulation
- ›TrkB receptor activation
- ›Dopaminergic & serotonergic modulation
- ›Acetylcholinesterase inhibition
- ›CREB phosphorylation enhancement
Primary Research Areas
- ›Neuroprotection
- ›Stroke recovery
- ›Cognitive enhancement
- ›Attention disorders
- ›Neurotrophic factor research
Key Research Findings
- Upregulated BDNF by 1.4–3x in rat hippocampus after intranasal administration
- Demonstrated neuroprotective effects in rat MCAO stroke model
- Improved spatial memory in Morris water maze tests
Research Overview
A heptapeptide derived from the ACTH(4-10) fragment, developed at the Institute of Molecular Genetics. Unlike the parent ACTH molecule, Semax lacks corticotropic and steroidogenic activity, instead focusing on nootropic and neuroprotective pathways.
Origin: Synthetic analog of ACTH(4-10) fragment with Pro-Gly-Pro extension
Mechanism of Action
- BDNF & NGF upregulation
- TrkB receptor activation
- Dopaminergic & serotonergic modulation
- Acetylcholinesterase inhibition
- CREB phosphorylation enhancement
Primary Research Areas
- Neuroprotection
- Stroke recovery
- Cognitive enhancement
- Attention disorders
- Neurotrophic factor research
Key Published Findings
- Upregulated BDNF by 1.4–3x in rat hippocampus after intranasal administration
- Demonstrated neuroprotective effects in rat MCAO stroke model
- Improved spatial memory in Morris water maze tests
Research Protocol
Commonly studied routes: Intranasal, Subcutaneous. Reconstitute with bacteriostatic water. Consult published literature for dose ranges; use our Peptide Calculator for volumetric preparation.
Storage
-20°C lyophilized
Important Notice
Semax (10mg) is supplied for laboratory research use only (RUO). Not for human or veterinary use, diagnostics, or therapeutics.
Spécifications de recherche
Synthesized for strict analytical consistency. Verified via HPLC/MS.Voir les standards qualité →
Manipulation et stockage
- Store lyophilized at -20°C
- Protect from light and moisture
- Use sterile bacteriostatic water for reconstitution
- Minimize freeze-thaw cycles
- Solubility: Soluble in water